10:40-12:00 Morning Sessions 2
Session C:
Chair: Yoni Haitin (TAU) and Reuven Wiener (HUJI)
“Structural Biology”
10:40-11:00 Moshe Giladi (TAU) Allosteric Regulation of Sodium-Calcium Exchangers: Insights From Hybrid Structural Biology Studies
11:00-11:20 Yoni Haitin (TAU) Structure and Oligomerization of Chloride Intracellular Channels(?) – A Developing Story…
11:20-11:40 Yarden Opatowsky (BIU) Slit-Robo in the Crosshairs, A Structural Study
11:40-12:00 Reuven Wiener (HUJI) Molecular Mechanisms of Protein Modification by UFM1
Session D:
Chair: Shai Berlin (Technion)
“Optogenetics: Remote Control Tools in Biological Systems“
10:40-11:00 Pierre Paoleti (PSL, Paris) Illuminating glutamate receptor structure and function
11:00-11:20 Amit Gruber (Technion) Optogenetic Control of Human Induced Pluripotent Stem Cell Derived Cadiac Tissues
11:20-11:40 Ilan Lampl (WIS) Optogenetic studies of correlation and decorrelation mechanisms in the barrel cortex
11:40-12:00 Shai Berlin (Technion) Two-photon compatible light-gated NMDARs
Session E:
Chair: Rena Yarom – Young Investigator Competition
In collaboration with the Israeli Section of the ISHR
10:40-10:55 La-Paz Levin-Kotler (102) Overexpression of the longevity gene SIRT6 in myeloid cells reduced left ventricular hypertrophy after myocardial infraction
10:55-11:10 Idit Goldfracht (116) Functional comparison of engineered atrial and ventricular heart tissues derived from human pluripotent stem cells
11:10-11:25 Michael Mutlak (114) Extracellular regulated kinases protects against maladaptive cardiac hypertrophy following chronic pressure overload
11:25-11:40 Wesam Mulla (118) QT interval and ventricular refractory period measurements indicate absence of typical rate-dependence in conscious freely moving rodents
11:40-11:55 Binyamin Eisen (106) Functional abnormalities in induced pluripotent stem cell-derived cardiomyocytes generated from a Duchenne muscular dystrophy patient and a female manifesting carrier
11:55-12:10 Roy Kalfon (115) The role of dual loss-of-function mutations in the bZIP-repressor proteins, ATF3 and JDP2, in cardiac remodeling and hypertrophy